Contributors: Jacob B. Hunter, Reid C. Thompson and David S. Haynes
Superior semicircular canal dehiscence (SCD) is a condition in which the bone overlying the superior semicircular canal is absent. The clinical presentation of SCD is highly variable and may include both auditory and vestibular manifestations. The more common symptoms include autophony, sound or pressure induced vertigo, hypersensitivity to sound, and low frequency conductive hearing loss. Repair can be accomplished via either transmastoid or middle fossa approaches, with numerous materials used to either plug or resurface the canal. Herein, we describe our resurfacing technique using a loose areolar tissue-bone pâté-loose areolar tissue sandwich through a middle fossa approach.
Patients with symptomatic SCD not controlled with conservative measures are candidates for repair.
Bleeding disorders should be optimized prior to surgery and patients should hold all anticoagulant medications for at least 1 week prior to surgery.
The patient is positioned in the park bench position and the head is placed in a Mayfield skull clamp.
The diagnosis of SCD is made radiographically, therefore all patients should undergo fine cut temporal bone computed tomography (CT) imaging. In addition, a complete history, physical examination and routine audiometric testing should be performed. While balance function testing is not required, we recommend such testing particularly in patients with vestibular complaints. Vestibular evoked myogenic potentials (VEMPs) can aid in the diagnosis of SCD.
In the coronal plane, the superior semicircular canal is usually located at the level of the posterior external auditory canal. Along the middle fossa floor, the superior semicircular canal is oriented perpendicularly to the posterior petrous ridge. Although the arcuate eminence corresponds to the location of the superior semicircular canal in the majority of patients, this relationship is not absolute. Furthermore, in patients with SCD, the arcuate eminence will likely not be as prominent. In relation to the lateral cortex of the temporal squama, the superior semicircular canal lies roughly 2.5 cm medially.
Risks include bleeding, infection, hearing loss, spinal fluid leak, dizziness, facial nerve weakness, change in taste, aphasia, stroke, and death.
Risks include bleeding, infection, hearing loss, spinal fluid leak, dizziness, facial nerve weakness, change in taste, aphasia, stroke, and death.
N/A
N/A
Review Fascia with Bone Pate Resurfacing Technique for Repair of Superior Semicircular Canal Dehiscence. Cancel reply
Related Videos
This video shows a transotic approach for a cochlear schwannoma in a 59-year-old female who presented with sudden sensorineural hearing loss in her left three years prior. She was found to have subsequent growth of her tumor on imaging and elected to undergo surgery. The transotic approach is a valuable approach within the armamentarium of a skull base team and differs from the transcochlear approach in the handling of the facial nerve. Techniques for ear canal overclosure, eustachian tube packing and mastoid obliteration are also highlighted.
Here, we have a 39 yrs old female with complaints of noisy breathing for last two years post thyroidectomy. Flexible laryngoscopy confirmed bilateral vocal cord paralysis. She was planned for coblation assisted cordectomy.
Patient was taken up for procedure under general anaesthesia. She also started having stridor after induction. Nasopharyngeal intubation with spontaneous breathing technique was used. Entropy leads were placed over forehead to monitor the depth of anaesthesia. Tube position was confirmed with endoscopic view and Benjamin lindohlm laryngoscope was suspended. As the patient was spontaneously breathing, the stridor became more prominent, with stable vitals and the procedure was continued. The vocal cord retractor was fixed and coblation wand was then used with 7:3 settings for ablation and coagulation respectively. The surgical limits were-anteriorly the junction between ant 2/3 and post 1/3 of the vocal cord, posteriorly just anterior to the vocal process of arytenoid to prevent cartilage exposure and post operative granulations. Superely till the ventricle and inferioly till the medial most surface of the subglottis. Laterally approx. 5 mm depth was defined to prevent injury to the superior laryngeal artery branch and further bleeding. Once the final airway was achieved , the topical lignocaine was used to prevent laryngeal spasm post extubation.
The patient was shifted to the ward without oxygen, the voice was assessed on post op day 2.
Patient was called for follow up on post op day 14th and good voice outcomes were achieved.
So lets have a look on some tips & tricks for the safe procedure—–
Nasopharyngeal insufflation technique with entropy monitor will give adequate and safe surgical field
2. Appropriate exposure will help you to delineate the surgical margins
3. Topical anaesthesia before and after the procedure will prevent sudden laryngeal spasm
4. Firm holding of coblation device will help to prevent injury to surrounding structures like anterior 2/3 vocal cord, opposite side vocal cord, medial surface of vocal cord or aryteroid posteriorly
5. Do not ablate more laterally to prevent bleeding, if at all it happens, use patties or coagulation switch for hemostasis.
6. And at the end of the procedure ,use catheter suction to suck out blood clots or saline from the airway if any….
To Conclude-Coblation Assisted Cordectomy( CAC) can be performed safely with good outcomes in case of bilateral vocal cord paralysis using tubeless anesthesia technique without tracheostomy !
We present the harvest of a osteocutaneous fibula free flap for head and neck reconstruction performed at the University of Cincinnati. This reconstructive technique has a wide variety of implications but has found greatest utility in the reconstruction of mandibular defects.
Review Fascia with Bone Pate Resurfacing Technique for Repair of Superior Semicircular Canal Dehiscence.